By Suresh Kotagal, Alma R. Bicknese, Marthand Eswara (auth.), Roger N. Rosenberg MD (eds.)
The newly revised, entire, four-color Atlas of medical Neurology is greatly up to date as regards to present literature. the hot 3rd variation organizes the highest-quality pictures from the sphere of scientific neurology, together with scientific syndromes, for neurologists and clinicians. The atlas highlights the big strides being made within the biologic knowing of neurologic ailment, masking developmental and genetic illnesses, neuroendocrine problems, severe care neurology, cerebrovascular ailment, dementias, behavioral neurology, neuro-oncology, circulation problems, epilepsy, neuromuscular illnesses, infectious illnesses, neuroimmunology, neurotoxic issues, and headache. This quintessential assortment comprises over 1300 pictures together with algorithms, tables, charts, and schematic drawings.
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Bicknese AR, Sheppard AM, O’Leary DDM, Pearlman AL: Thalamocortical afferents extend along a chondroitin sulfate proteoglycan-enriched pathway coincident with the neocortical subplate and distinct from the efferent pathway. J Neuroscii 1994, 14:3500–3510. 32. Rakic P: Mode of cell migration to the superﬁcial layers of fetal monkey neocortex. J Comp Neuroll 1972, 145:61–83. 33. Dobyns WB, Gilbert EF, Opitz JM: Further comments on the lissencephaly syndromes. Am J Med Genett 1985, 22:197–211. 54.
Most patients develop evolving cutaneous lesions (stage 1, bullae; stage 2, verrucae) in the ﬁrst year of life, although the cutaneous manifestations may appear at single or multiple stages at birth with accompanying neurologic symptoms. Overall systemic involvement occurs in 80% of patients who manifest cutaneous lesions. The female to male ratio is 35 to 1. X-linked dominant inheritance is the likely basis for the predominance of affected girls and presumed lethality in affected boys. In our own experience, fathers with cutaneous lesions of IP had no neurologic symptoms, and the literature indicates that surviving affected males have no greater degree of neurologic involvement.
JAMA 1993, 270:2838–2842. 3. Guttman RH, Collins FS: Recent progress toward understanding the molecular biology of von Recklinghausen’s neuroﬁbromatosis. Ann Neuroll 1992, 31:555–561. 4. Use of folic acid for prevention of spina biﬁda and other neural tube defects: 1983–1991. MMWR Morb Mortal Wkly Rep 1991, 40:513–516. 5. Shaer CM, Chescheir N, Schulkin J: Myelomeningocele: a review of the epidemiology, genetics, risk factors for conception, prenatal diagnosis, and prognosis for affected individuals.